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We reconstructed metabolic models to investigate the dependency of the P

in 2008, patients with NASH possess failure to mobilize an XBP1-mediated increase in splicing in EDEM1, which promotes the proteomic degradation of ubiquitinated proteins

It significantly reduced the HbA1c and improved coronary flow reserve (CFR) and left ventricular ejection fraction (LVEF) after ATP (adenosine triphosphate) infusion

(2024, June 1)